Tuesday, September 01, 2009

Complete GI Test en Metametrix

Remember that this is just a post of my blog, and it evolves, so to see the full story go to: www.pochoams.blogspot.com (English) or www.sfc-tratamiento.blogspot.com (Spanish)
My current doc: Josepa Rigau Av Catalunya, 12, 3º, 1ª 43002 Tarragona Spain +34977220358 (I do recommend! hoeopathy and biological medicine, significant improvement)
My previous docs: De Meirleir (www.redlabs.be), Dra Quintana (CMD), (Lots of medication, antibiotics etc... no significant improvement)

Stephanie E. Wickham
Consumer / Sales Liaison
sewickham@metametrix.com
www.metametrix.com
Tel: 16786382910

This is the address to take a complete test and fecal flora in Metrametrix.
PCR using a technique that is not wrong or no bug leaves out of the picture, so whatever you have in the intestine comes out in the photo. What I usually do in Holland in the ELN (European Laboratory of Nutrients) but as I no longer live there, but in Madrid, then I'll do it in this that is more advanced at the PCR, and is a good way to test my flora and if there is bug or not within my bowels. In theory would not necessarily have, but my trouble lately, so I want to look ... contare you and then I just get the kit

Of course you pay the shipping back and forth ... have to write to such Stephanie with the details of your credit card to send you the kit costs 465 $ This test is not cheap.

Spoke of a recent test I have made in the USA from my stool in the laboratory Metametrix
They use an advanced PCR technique to detect especially in terms of flora and pathogens.

1) Stresses a very low level of pancreatic elastase 1, which often bears a strong correlation with pancreatic insufficiency. The causes include: hipocloridia, pancreatic insufficiency, chronic pancreatitis, diabetes, fibrosis quitica, load ... The best treatment is: Betaine HCl, pancreatic enzymes, or digestive herbs (ginger or mint). Taurine, bile salts (especially if constipation or high triglycerides). eat slowly relaxed, and caring for the regulation of diabetes.

On this occasion neither triglycerides nor any other marker would indicate poor absorption of fats, but remember that in another analysis that I did in the past in Holland if I went out malabsorption of fat, according to the estatorrea noting the low level pancreatic elastase.

2) A low level of fecal IgA secretion denotes a low reactivity of the immune system to the presence of antigens from bacteria, fungi or microbes. This may be due to stress or malnutrition, or an intestinal dysbiosis. Proper treatment would be: take pancreatic enzymes, betaine HCl, or digestive herbs. Support the intestinal mucosa with glutamine, probiotics or mucosa compositum. Take bifidus and S. Boulardii, colostrum, immunoglobulins, omega, zinc, reduce stress and strengthen the immune system.

3) The short-chain fatty acids essential for a good balance of intestinal flora, are very upset for me, especially the n-Butyrate and Butyrate that handle normalize the activity of colonic epithelial cells and prevent colorectal cancer and colitis. The best treatment would be: Take probiotics, take fiber: psyllium, inulin, oligofructose, oat bran, betaglucan, arabinogalactan, increase consumption of fruits and vegetables or supplements butyrate enemas of butyrate with capsule whole.

4) The index of adiposity have been altered, because the DNA test detects a high level of Firmicutes (Lactobacillus sp. and Mycoplasma sp.) and low level of Bacteroides (Bacteroides sp. and Prevotella sp.). This abnormality of the edges may be associated with increased caloric extraction from food. Proper treatment would: eliminate opportunistic bacteria such as bacilli, taking bifidus and S. Boulardii (Ultra-levure), reduce intake of refined carbohydrates, Balancing all gastrointestinal imbalances.

5) There is a protozoa present, taxonomy unavailable, which suggests that this is a protozoa that is not known and could be passing without symptoms, or conversely could be a rare protozoan protozoa belonging to the universe because their DNA has been detected, and this is not a human parasite. Could come from a pet, or food and that way, or however be a rare species if it is causing symptoms. We must assess whether they have traveled abroad, or if other parameters of the test that could support the infection theory as it may be a high level of lactoferrin, which is not my case. However if in my case pancreatic insufficiency proven by the low levels of elastase 1, a low level of secretion of IgA, and an imbalance of short chain fatty acids, if point to intestinal dysbiosis, on the other hand this found by the urine test from Meirlaen. If you suspect this is responsible for the symptoms can be treated with a broad spectrum antiparasitic, or taking a botanical treatment.

This time there is an overgrowth of fungus on this occasion, in the past if there was an overgrowth of candida (tricosporon cutaneum).

Beneficial bacteria have come into balance, because the last time he had no just or Lactobacillus bifidus as well as Escherichia coli. On this occasion, after having taken mutaflor, all are in range, and whether there is an excess of mycoplasma and lactobacilli, but as they are beneficial bacteria are not considered an abnormality, unless many of them are high at a time. Curiously, according to The excess H2S Meirlaen urine is caused by an overgrowth of Streptococcus, Enterococcus and Prevotella, however Prevotella my levels are normal, however not reflect the level of Streptococcus and Enterococcus in this analysis.

As pathogens detected
there are 4, but none of them in relevant quantities, which need not be.
Helicobacter pylori
Clostridium difficile
E.H.E. coli
Campylobacter sp.

Reference: http://www.metametrix.com/DirectoryOfServices/pdf/pdf_guide_GIf-Interpretive-Guide.pdf

In short:

Well ... what to do in my case is pretty clear in the page metametrix, which incidentally coincides rather with what he sends me or sent me in the past, my Doctor Josep Rigau:

"Eat slowly relaxed
-Pancreatic Enzymes
-Betaine HCI
-L-Glutamine
-Mucosa Compositum
-Probiotic (Bifidus and Ultra-levure S. boulardii)
-Omegas
-Zinc
-Reduce stress and increase consumption of fruits and vegetables and reduce intake of refined carbohydrates.
-Taurine
-Take fiber: Psyllium

Other things however are new and will discuss with her:

-Colostrum (transfer factor)
-Herbal digestive (Ginger or Peppermint)
-Immunoglobulin
-Bile Salts
-Inulin
-Supplement with whole capsule butyrate
"Oligofructose
Oat bran -
-Betaglucan
-Arabinogalactan

The facts about IBS

* Irritable Bowel Syndrome remains a controversial disease because of its lack of consistent symptoms. It is diagnosed only after ruling out other causes such as parasitic infections, lactose intolerance, small intestinal bacterial overgrowth and coeliac disease.

* The symptoms generally include bloating, abdominal pain and discomfort and a change in bowel habits.

* Gastrointestinal infections can be a catalyst for the onset of IBS, with sufferers of infections six times more likely to develop IBS.

* Certain foods appear more likely to prompt IBS symptoms, including dairy and wheat, which, along with the similarity of symptoms, is one of the reasons that coeliac disease might be mistaken for IBS. IBS sufferers should request testing for coeliac disease.

* In research IBS has repeatedly been linked to the mental and nervous state, with stress, depression and chronic fatigue syndrome being prevalent among IBS sufferers. Current theories increasingly emphasise the “Gut-brain axis”, which connects the actions of the gut with psychological factors.

* Treatments include those related to the gut-brain axis including gut-focused hypnotherapy and cognitive behavioural therapy, dietary changes such as excluding wheats and increasing fibre intake, probiotics and an increase in exercise. However, the effectiveness of these varies between patients and it is usually by trial and error that an appropriate treatment will be found.

* A study released last month by the Mayo Clinic in Minnesota found that people with IBS were three times as likely to have a relative who also had the disorder, research that may prompt a search for an “IBS gene”.